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TMPO-AS1 enhanced CESC proliferation and migration by suppressing ferroptosis
Chondrocyte apoptosis in the pathogenesis of osteoarthritis
Primary glutamatergic deficit, likely originating from aberrant neuron-glia interactions, leads to an early GSH excess via diversion of GSH-glutamate metabolic pathways
Nature 403 , 4145 (2000)
[PubMed] [Google Scholar] 49.Sergio W
GLS1 is overexpressed in various cancer cells, and this phenotype is associated with a higher disease stage and a poor prognosis 10
