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Loss of PRMT7 in the heart reduces symmetrical dimethylation of -catenin, enhances Wnt--catenin signaling, and promotes myocardial hypertrophy

Eur J Pharmacol 932:175208

found increased expression of a fibrosis marker, fibroblast activating protein (FAP), and markers of oxidative stress, inflammation, and autophagy in obese patients compared to non-obese individuals, as stated prior in this review [11]

Many transcription factors and signaling molecules have critical cysteine residues that can be oxidized and this is an important mechanism whereby reactive oxygen and nitrogen species (ROS and RNS) regulate protein function and cell signaling that can be modulated by GSH [13,15]

PMC Zosky GR, Berry LJ, Elliot JG, et al

Oxidative stress, which results from the imbalance between the production and neutralization of ROS, is also reported to be involved in a large number of pathological states, such as Alzheimers disease [30], Parkinsons disease [31], atherosclerosis [32], heart failure [33], myocardial infarction [34], and hepatic encephalopathy [35]