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Hippocampal volume in adult burn patients with and without posttraumatic stress disorder

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Proteomic analysis revealed that while majority of mitochondrial protein were unaffected upon exposure to DA, rapid loss of subset mitochondrial proteins including NADH dehydrogenase (75 kDa) and NADH oxidoreductase (30 kDa) subset of complex I, mitochondrial kinase, mortalin/GRP75, mitofilin, HSP60, peroxiredoxin 3 and voltage-dependent anion 2 were observed, many of which play critical role in tight regulation of mitochondrial homeostasis [345]