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The peptide's activation of ERK1/2 and downstream transcription factors including Egr-1 drives expression of collagen synthesis genes while its enhancement of cellular migration ensures efficient fibroblast recruitment to sites requiring matrix deposition, creating optimal conditions for rapid yet organized tissue reconstruction

Here, the authors report on a mitochondrial targeting nanomotor with self-feedback mechanisms to trigger the mineralisation of mitochondria which together with curcumin, inhibits glioblastoma progression.

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