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Hence, although these sleep deprivation-induced immune-mediated mechanisms in cancer warrant further confirmation in humans, the importance of the immune function in the anti-tumor host defense is well recognized 131 , thus suggesting that the impaired immune response after sleep deprivation may represent a plausible mediator of the associated increased risk for cancer as described in animal models and in humans

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The success of this method solely depends on the expression, solubility and stability of the desired protein